New research finds that the deletion of the ataxia-telangiectasia group D-complementing (Atdc) gene, whose human homolog is up-regulated in the majority of pancreatic cancer cases, completely prevents the development of tumors in mice.
The work is published online in the journal Genes & Development in a paper titled, “ATDC is required for the initiation of KRAS-induced pancreatic tumorigenesis.”
